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A number of clinical observations suggest that Down patients may have a functional folate deficiency despite having normal plasma levels of folate and vitamin B 12 [13]

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These antibodies contribute to glandular injury via complement-dependent and antibody-dependent cell-mediated cytotoxicity, and promote the release of cytokines such as IL-6, IL-1, and IL-8, which enhance lymphocyte accumulation and the local inflammatory response

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